The south of England and the Scottish Highlands have been earmarked as high risk areas for Lyme disease.
The human transmission of Lyme disease starts from ticks, which are external insects that feed on blood sucked from humans and animals. The tick becomes infected by pulling bacteria of the genus Borrelia from the infected host (animal, human). Borrelia sp. exists in three morphological forms which allow them to withstand and survive changing, and even hostile, environments. These are: active form (spirochetes), and latent forms (rounded forms and biofilm).
Scientists at the Dr. Rath Research Institute have tested 45 natural compounds against two species of Borrelia: Borrelia burgdorferi sensu stricto (the pathogen causing Lyme disease in the USA) and Borrelia garinii (the pathogen causing Lyme disease in Europe). Their results showed that all tested compounds inhibited the bacterial growth of spirochetes. The most effective substances that induced the death of latent rounded forms of Borrelia were cis-2- decenoic acid, rosmarinic acid, baicalein, monolaurin, luteolin, and kelp (iodine). Five of the compounds, baicalein, luteolin, monolaurin, cis-2-decenoic acid, and kelp (iodine), could also reduce biofilm-like colonies formed by Borrelia burgdorferi, although only baicalein and monolaurin could reduce biofilm formation by Borrelia garinii.