These new NIH studies confirm that elevated Lipoprotein(a), or Lp(a), is a significant and independent driver of cardiovascular risk, even in patients already receiving standard treatments. The researchers found that individuals with Lp(a) levels of 175 nmol/L or higher faced notably increased risks of major adverse cardiovascular events.
In recognizing the importance of Lp(a), the new NIH data shows once again that Dr. Rath was right. In a groundbreaking study published in 2015, scientists from the Dr. Rath Research Institute proved that cardiovascular disease is an early form of the vitamin C deficiency disease, scurvy. This study used transgenic mice that mimic human metabolism with regard to two key genetic features: an inability to produce vitamin C, and the ability to produce Lp(a). When given a diet containing an insufficient amount of vitamin C, the mice were found to deposit Lp(a) in their vascular walls and develop atherosclerosis.
The degree to which these mice developed cardiovascular disease was found to be inversely related to their intake of vitamin C. Those with the highest intake of vitamin C had the lowest depositions of Lp(a) in their arteries and the least atherosclerotic lesion development. As such, while NIH’s recognition of the risk of raised Lp(a) levels is clearly a welcome development, a full understanding of the biological mechanism behind the molecule’s connection with cardiovascular disease can only be obtained through factoring in Dr. Rath’s discovery of the association between raised levels of it and a low intake of vitamin C.
To learn how heart attacks, strokes, high blood pressure, diabetes, high cholesterol, and other cardiovascular conditions can be prevented naturally, read Dr. Rath’s classic book: Why Animals Don’t Get Heart Attacks … But People Do!
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May 1, 2026New Studies Link Lipoprotein(a) to Elevated Cardiovascular Risk
News
New data analyzing more than 20,000 patients from three major United States National Institutes of Health (NIH) studies show that elevated levels of the molecule Lipoprotein(a) are associated with cardiovascular risk and warrant aggressive risk reduction.
[Source: medicalxpress.com]
[Image source: freepik.com]
Comment
These new NIH studies confirm that elevated Lipoprotein(a), or Lp(a), is a significant and independent driver of cardiovascular risk, even in patients already receiving standard treatments. The researchers found that individuals with Lp(a) levels of 175 nmol/L or higher faced notably increased risks of major adverse cardiovascular events.
In recognizing the importance of Lp(a), the new NIH data shows once again that Dr. Rath was right. In a groundbreaking study published in 2015, scientists from the Dr. Rath Research Institute proved that cardiovascular disease is an early form of the vitamin C deficiency disease, scurvy. This study used transgenic mice that mimic human metabolism with regard to two key genetic features: an inability to produce vitamin C, and the ability to produce Lp(a). When given a diet containing an insufficient amount of vitamin C, the mice were found to deposit Lp(a) in their vascular walls and develop atherosclerosis.
The degree to which these mice developed cardiovascular disease was found to be inversely related to their intake of vitamin C. Those with the highest intake of vitamin C had the lowest depositions of Lp(a) in their arteries and the least atherosclerotic lesion development. As such, while NIH’s recognition of the risk of raised Lp(a) levels is clearly a welcome development, a full understanding of the biological mechanism behind the molecule’s connection with cardiovascular disease can only be obtained through factoring in Dr. Rath’s discovery of the association between raised levels of it and a low intake of vitamin C.
To learn how heart attacks, strokes, high blood pressure, diabetes, high cholesterol, and other cardiovascular conditions can be prevented naturally, read Dr. Rath’s classic book: Why Animals Don’t Get Heart Attacks … But People Do!
Dr. Rath Health Foundation
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