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New Coronavirus Variant Spreading In New York


“A new form of the coronavirus spreading rapidly in New York City carries a worrisome mutation that may weaken the effectiveness of vaccines, two teams of researchers have found.” [Source:]


As Dr. Rath described in his recent letter to world leaders, the ongoing mutations of the coronavirus require the immediate reorienting of medical research and global health strategies. Despite claims by some vaccine manufacturers that their current products would ‘work’ against these mutations, there is currently no definitive scientific proof – in the form of large-scale peer-reviewed clinical studies – that existing vaccines are effective in patients infected with them.

Constant mutations of viruses – a slight restructuring of their architecture (genetic sequence) – are not the exception in the viral world, but the rule. Virus mutations are not just random events, but a survival mechanism that allows them to escape the immune defenses of the living beings (hosts) they infect, including humans.

The coronavirus is mutating at a high rate, with several mutations having already occurred within one year of the current pandemic starting. Examples include those found in the United Kingdom, South Africa, Brazil, and other countries. These initial mutations are just the ‘tip of the iceberg’, however, as every new mutation potentially threatens humankind with a new pandemic.

Even if it should turn out that existing vaccines offer some protection against one or another current mutation, future mutations of the coronavirus will almost certainly evade such protection. The predictable consequence is that with every new mutation, the world will be at risk of being exposed to a crisis similar to that which it has been experiencing since early 2020. A strategy based on vaccines alone therefore cannot be the solution to the current pandemic.

To read how groundbreaking studies carried out by scientists at the Dr. Rath Research Institute have shown that synergistic combinations of micronutrients can block the interaction between the binding site of the coronavirus ‘spike’ and its specific cellular entry door into human cells, see the press releases we published in July and August 2020.